 Centrophenoxine ‘cleans out’ the cells in your body and brain, improving their function and their longevity
Centrophenoxine has long being known to be extremely effective at reducing lipofuscin levels. Lipofuscin describes the potassium build up in the brain, heart, lung and skin cells. This biochemical clutter accumulates over a lifetime, sometimes reaching 30% of the cell volume in aged animals.
The more lipofuscin a cell accumulates, the less functional it becomes, and once a critical threshold has been reached the cell may die. Both human and animal studies have shown that low lipofuscin levels correlate with healthy cellular function, while high lipofuscin accompanies poor cellular health.
Through a variety of animal experiments measuring learning abilities and memory, aged animals whose high lipofuscin levels were reduced by Centrophenoxine, also had their memory and learning abilities restored to a level similar to healthy young animals, while untreated high lipofuscin control animals did not.
Centrophenoxine also generally improves brain function. It enhances neuronal glucose (the chief brain fuel) and oxygen uptake, while increasing carbon dioxide production. In addition, Centrophenoxine raises neuronal RNA (derived from DNA in the cell nucleus) which enables neurons to form proteins which help encode memory, as well as repair cell damage.
Centrophenoxine reverses the age-related drop in RNA and protein production. One study with 76 healthy elderly, who suffered from significant intellectual deterioration, found that Centrophenoxine increased storage of new information into long term memory, while also increasing vigilance and alertness, after only several weeks of treatment.
The drug Centrophenoxine has played a central role in many antiaging experiments. Scientific experiments have proven the free radical scavenging properties of DMAE. It should be stressed that this part of Centrophenoxine is actually incorporated into the brain cell membrane. The presence of DMAE in the brain cell membrane provides increased protection against damaging free radical attacks. The present findings offer strong evidence supporting the observation that vital membranes are protected during continuous treatment with Centrophenoxine. The other compound in Centrophenoxine also is a powerful free radical scavenger, although probably not as important as the DMAE portion. The combined effect of both parts of the drug prevents free radical induced aging damage and improves the body's defence against free radical attack.
Many scientific experiments have revealed a considerable age-dependent drop in levels of the chemical RNA in the brain. RNA is essential for the body to make protein. Treatment with Centrophenoxine is able to reverse this decrease in production of RNA. Since the increased rate of RNA production allows the cell to carry out protein synthesis at a higher rate, the effects of Centrophenoxine may also involve an increased rate of protein turnover. This increased rate of protein turnover is important because it is related to all the beneficial antiaging effects of Centrophenoxine.
Various drugs have been developed to treat senile dementia, and Centrophenoxine has been found to be useful. Regular use of Centrophenoxine offers you the chance to prevent mental deterioration. Memory performance is improved in both the normal elderly and those with dementia following treatment with Centrophenoxine . The most obvious change in the nerve cells of elderly individuals is the appearance and increase in the amount of lipofuscin pigment. Experimental studies have shown that Centrophenoxine treatment can reduce the amount of lipofuscin in nerve cells. This suggests that the beneficial effects on mental function in demented patients is partly due to removal of age pigment from nerve cells. Many scientists believe that benefits also are produced by the improvements in cell metabolism that Centrophenoxine produces.
Senile dementia is related to changes in the part of the brain known as the hippocampus. The nerve cells of the hippocampus are damaged as part of the aging process; however, in senile dementia these damaging changes are much worse. Similar changes also may occur in the rest of the central nervous system. This extra damage indicates a further decline in metabolic activity in affected nerve cells. A very important effect of Centrophenoxine is the stimulation of uptake of glucose by the brain. This can help to make up for the damage caused to the hippocampus by aging and senile dementia. The beneficial effects on mental performance seen in older people following regular use of Centrophenoxine are due to a real improvement in the efficiency of the workings of vital cells. Nerve cells in particular work much better under the influence of Centrophenoxine..
An attempt to explain exactly how Centrophenoxine works has been made using the "membrane theory of aging". The "membrane theory of aging" attributes a primary role in aging to a decrease in the ability of the mineral potassium to get into and out of cells. This causes the level of potassium in the cell to rise leading to a reduction in essential protein production. the rise in potassium levels is due to free-radical induced cross-linking in the cell membrane. the "membrane theory of aging" as outlined above suggest that the cells of the body gradually lose their ability to eliminate damaged components i.e.. lipofuscin builds up within the cells. Centrophenoxine slows down the rate of accumulation of age deposits (lipofuscin) in brain cells and it also removes deposits that have already accumulated in aged brains, is incorporated into the membranes of brain cells, where it protects against the cross-linking effects of free-radicals. The drug is able to increase the storage of new information in long-term memory and increase mental alertness.
Dosage: Elderly people with significant intellectual decline/ may need three to six 250mg tablets per day taken preferably with breakfast and lunch, in order to avoid insomnia. Healthy younger people may need only one or two 250mg tablets daily with breakfast or lunch. To avoid any slight acetylcholine excess occurring, it is wise take occasional breaks from dosage (say, one day a week).
Safety: There is virtually no toxicity associated with Centrophenoxine in therapeutic doses. People with the following medical conditions should not take Centrophenoxine: severe high blood pressure, convulsive disorders including epilepsy, pregnant and lactating women.
Persons taking prescription medication should consult a physician before using this product. Long-term use of this product should be supervised and monitored by a health care professional.
Disclaimer: This product and its statements have not been evaluated by the FDA. This product is not intended to treat, cure or prevent any disease. The above information is provided under the supplying company's terms and conditions and should not replace the advice of your personal physician.
Restrictions: This product is not available for shipment to the UK
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